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Structure 23 , 14261436 (2015)

It may also boost nitric oxide production, which improves blood flow and lowers the risk of high blood pressure

Cancer 71 KumarendranB.OReillyM

Differential pathophysiological mechanisms between CHS and EHS While CHS and EHS share core pathogenic pathways (e.g., hyperthermia, inflammation, cell death), their mechanistic features differ significantly due to distinct patient demographics and triggers: Inflammatory kinetics EHS (predominant in young, active individuals) is characterized by rapid, exertion-amplified inflammatory responses