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Taken together, we provide genetic evidence that glutathione plays an important role in AITC-induced growth inhibition, although further studies need to be conducted to reveal the underlying mechanisms

Mechanistically, this may involve: Increased adiponectin levels Decreased leptin concentrations Enhanced fatty acid oxidation Reduced hepatic steatosis Improved lipid clearance The glucagon receptor component appears to drive much of the enhanced liver fat reduction observed in clinical trials, distinguishing retatrutide from GLP-1only therapies

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