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The VPA-exposed group exhibited activated microglia characterized by hypertrophic morphology with significantly elevated Iba1 fluorescence intensity compared to the control group (P 0.05)

51 Following this translocation, microbial-associated molecular patterns and damage-associated molecular patterns from stressed and damaged cells can enter the liver through the portal vein, activating immune and parenchymal cells on the surfaces of toll-like and nucleotide-binding oligomerization domain-like receptors, triggering inflammatory responses, promoting hepatic fat accumulation, and accelerating liver fibrosis

In most cases, however, the underlying peptide sequence is intended to remain the same

pylori is present